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bioinformatics. The role will be responsible for developing and characterising human dorsal root ganglia cultures to benchmark the newly developed iPSC derived organoid model systems. This will include processing
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ecomorphological outcomes significantly parallel. As a next step, it is vital to dig more deeply into the molecular mechanisms driving these patterns. This project will examine replicate divergences into specialist
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of multiple medicines across cellular mechanisms in parallel, ensuring accelerated assessment of existing and new therapies. Improving our understanding of the cellular basis of disease will help bridge the gap
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bioinformatics. The role will be responsible for developing and characterising human dorsal root ganglia cultures to benchmark the newly developed iPSC derived organoid model systems. This will include processing
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metabolic disease, including type 2 diabetes (T2D), Non-alcoholic fatty liver disease (NAFLD) and obesity, collectively referred to as metabolic syndrome (MetS), has occurred in parallel with a global decline
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controls almost every cellular process in humans, including targeting poorly folded proteins for destruction to prevent them harming the cell. When ubiquitin signalling goes wrong, many different disease
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cells, most notably immune cells. This project will investigate how cells coordinate these parallel nutrient scavenging approaches. The aim is to uncover the molecular mechanisms by which they talk
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biomaterials, including tuneable 2D and 3D culture systems, advanced microscopy, and polysome profiling, to study the impact of mechanical cues on ribosome function. In parallel, you will use bioinformatics