-
and Saeys teams. In this research project you will develop and apply algorithms to link clinical phenotypes of metastasis to molecular phenotypes in mouse models. It is known that metastases exhibit
-
the development of genetic autoinflammatory diseases. Using techniques in cell biology, molecular biology and biophysics you will investigate how loss of function of the RNA editing enzyme ADAR1 causes the human
-
lab first described that dominant mutations in YARS1 cause CMT (Jordanova et al., Nat Genetics 2006) and linked the enzyme’s non-canoncial functions to neurodegeneration. To gain better insights in
-
is to integrate genetics, cell biology, genomics, and bio-computing to unravel plant biological processes and to further translate this knowledge into value for society. Please visit us at