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that enable structural determination of native chromatin-associated complexes, we aim to elucidate the structural foundations of key biological events on eukaryotic chromosomes. The Arimura Lab provides a
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conception of the molecular biology of cells is going from the equivalent of grainy black-and-white to high-resolution technicolor. From deciphering the choreography of transcription complexes, chromosomes
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Division of Time 80% Research: Build, train, and evaluate cutting-edge AI models and multi-agent systems for conservation genomics; perform feature engineering using chromosome-level genome assemblies. 15
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that polyploidization disrupts dosage compensation in species with sex chromosomes. If verified, this theory could explain the rarity of polyploids in animals compared to plants. We will use Silene latifolia, a dioecious
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chromosome-level genome assemblies. 15% Writing: Contribute to scientific manuscripts, progress reports, and collaborative research efforts. 5% Professional Development. Requirements PhD Degree in
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of extra-chromosomal DNA in human cancers. The position is funded for initially two years (with options of extension) by the CRUK and NCI Cancer Grand Challenge to work on the evolutionary theory and
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biology techniques including PCR and cell culture will be essential. Experience of Next Generation Sequencing based protocols including ATAC-seq, ChIP-seq and Advanced Chromosome Conformation Capture
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Postdoctoral researcher in evolutionary genomics of sex chromosomes and sex-specific recombination in anurans andfor the validation of your doctoral research and, contains: A postdoctoral position is available
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crop improvement. The successful candidate will work on chromosome-scale genome assembly, gene annotation, structural variation discovery and development of genomics tools to accelerate breeding across
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Postdoctoral position to study Polo kinase and centrosome abnormalities in cancer and other diseases
centriole duplication, bipolar spindle formation, chromosome segregation, cell division, and proliferation. Dysregulation of Plk4/Plk1-dependent processes, by mutations in their associated cellular components