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at understanding the normal cellular trajectories of the brain, cutting-edge single-cell multi-omics approaches (sequencing and imaging based) to map cellular states and their transitions, pediatric neuro-oncology
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Assistant professor (post-doc) at the Laboratory of Nuclear Proteins at the Faculty of Biotechnology
of Wroclaw. Employment under the Sonata 17 project of National Science Center project, under the title “Identification of molecular mechanism underlying the phenotype development of Emery-Dreifuss muscular
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demonstrated accelerated tumor growth upon immunotherapy when cancer cell lost ATRX expression. Therefore, the study has potential to contribute to new biological insight on epigenetic mechanisms of resistance
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to elucidate the mechanisms by which ablation therapies influence the tumor microenvironment and immune response in pancreatic cancer. Key Responsibilities: Lead experiments investigating the tumor
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to understand mechanisms of dynein-2 transport, building on recent advances in the group (NSMB 2017 PMID 28394326; NSMB 2019 PMID 31451806; NSMB 2025 PMID 40730907; Cell 2022, PMID: 36462505). The research will
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Postdoctoral Research Associate - Developmental Neurobiology - Muscle-to-Brain Communication & Aging
the Department of Developmental Neurobiology at St. Jude Children's Research Hospital in Memphis, TN, USA. We use mice and Drosophila to decipher the conserved mechanisms of muscle aging and proteostasis, and how
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how immune mechanisms contribute to brain health disorders, using human stem-cell-derived models, multi-omic approaches, and clinically informed experimental designs. The work is embedded within a
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or mitochondrial dysfunction. Perform experiments utilizing molecular, genetic, cellular and imaging approaches in both rodent and cell models. Participate in the development and validation of rodent and cell models
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and applying theory to living systems on various scales, ranging from molecules to cells and tissues? Are you a theorist with strong expertise in statistical physics, soft/condensed matter theory
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strategies in CNS tumors by linking cellular states to functional vulnerabilities arising from their intrinsic molecular and phenotypic heterogeneity. The project will integrate single-cell and bulk multi