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of engineered tendon models Quantify and analyze inflammatory signaling in tendon fibroblasts Investigate ECM stiffness effects on cellular behavior and gene expression Develop fluorescent reporter systems
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ranges from reprogramming intracellular signaling of cells to modeling whole joint mechanics to understand and modify these systems at their respective length scales. Our research focuses on engineering
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how exercise protects the heart against heart failure. A growing interest surrounds common mechanisms affecting both the heart and the brain, and how the organs signal to each other in health and
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fellows, graduate students, research faculty, and medical/undergraduate students. Our work focuses on investigating the biology of DNA damage response signaling pathways in tumorigenesis and therapeutic
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Able to work independently and as part of a collaborative team Programming experience (e.g., LabVIEW, Matlab, etc.) Knowledge of signal processing and advanced statistics Work Locations Burlington
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meaningful information from noisy or ambiguous sensory signals? To what extent does neural plasticity in the auditory system occur via mechanisms dictated by classic theories of reinforcement learning, or does
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endonuclease assay, bacterial transformation and plasmid propagation, DNA cloning and point mutagenesis. Generation of CRISPR-Cas9-engineered models: Engineer cancer cells using the CRISPR-Cas9 technique
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meetings Required Qualifications* Highly motivated and enthusiastic individual with an MD/PhD or PhD background in cell and molecular biology, signal transduction, CRISPR gene editing, and bulk and scRNA