452 coding-"https:" "https:" "https:" "https:" "https:" "https:" "https:" "University of Essex" positions at University of Sheffield
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that the toxin induces DNA damage responses in cultured cells that activates a senescence tumour suppressor mechanism (https://doi.org/10.1038/s41467-019-12064-1). Cells undergoing toxin-induced senescence undergo
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application form using the following link: https://www.sheffield.ac.uk/postgraduate/phd/apply/applying All applicants should ensure that both references are uploaded onto their application as a decision will be
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instrumentation for materials formulation, processing, characterisation and performance assessment. More information on these facilities can be found at: https://www.sheffield.ac.uk/royce-institute https
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Matcher (https://www.sheffield.ac.uk/eee/people/academic-staff/stephen-matcher ) and the Human Parturition Research Group of Prof Dilly Anumba (https://www.sheffield.ac.uk/smph/people/clinical-medicine
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about this project. Funding Notes We welcome inquiries from: - applicants that have already secured PhD funding - self-funded applicants References https://microbialphysicsgroup.sites.sheffield.ac.uk
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interdisciplinary research. Please apply for this project using this link: https://www.sheffield.ac.uk/postgraduate/phd/apply/applying Funding Notes This is a self-funded project. Applicants must secure their own
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of technologies that are used in academia, industry and many related careers. Visit http://www.sheffield.ac.uk/sgs to learn more. Please apply for this project using this link: https://www.sheffield.ac.uk
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funding. References 1. Small-scale reconstruction in three-dimensional Kolmogorov flows using four-dimensional variational data assimilation (https://www.cambridge.org/core/journals/journal-of-fluid
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-funding, however, it other grant funding may arise such applications will also be considered. References For further reading see e.g., De Pontieu, Erdelyi and James, Nature 430, pages 536–539 (2004) https
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: Applications accepted all year round Details Mutation in the non-coding region of C9orf72 gene represents by far the most common genetic cause identified for amyotrophic lateral sclerosis (ALS), a rare, lethal