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) and ATTR (transthyretin) amyloidosis are the most common systemic forms, with distinct molecular origins but shared mechanisms of extracellular amyloid deposition and tissue damage. Our research program
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work with a team of scientists interested in (1) mechanisms of neurodegeneration, (2) cellular determinants of disease, (3) functional genomics, (3) the development of translational strategies in circuit
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interactions that underlie disease causation. Your job You will work with a team of scientists interested in (1) mechanisms of neurodegeneration, (2) cellular determinants of disease, (3) functional genomics, (3
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for Molecular Neurology are recruiting a postdoctoral researcher to establish next-generation humanized and engraftment models for studying peripheral neuroimmune mechanisms driving Parkinson’s disease. Building