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Start date: Autumn 2025 Duration: 3 years, with a 1-year extension negotiable Location: Department of Computer Science, Aalto University, Helsinki, Finland Apply: Applications reviewed on a rolling
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at the institute operate across multiple scales of biological resolution in the areas of Structural Biology, Molecular and Cell Biology, Developmental Biology, Genomics, and Evolutionary Biology. We seek an early
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experience during their PhD and postdoc periods. The selected group leaders are expected to initiate a new independent research program in one of FIMM’s research fields. Background FIMM is a leading molecular
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RESEARCHER / DOCTORAL RESEARCHER We are looking for a postdoctoral researcher to work on our projects that involve multiple sclerosis and myasthenia gravis. Also, applications for a PhD student position are
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at the institute operate across multiple scales of biological resolution in the areas of Structural Biology, Molecular and Cell Biology, Developmental Biology, Genomics, and Evolutionary Biology. We seek an early
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experience during their PhD and postdoc periods. The selected group leaders are expected to initiate a new independent research program in one of FIMM’s research fields. Background FIMM is a leading molecular
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group, established in 2018 at the University of Oslo, has recently expanded to the University of Helsinki. The group specializes in developing computational methods to understand the regulatory mechanisms
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experience in wet-lab and computational techniques. This position is intended for an MSc-level graduate planning to pursue a PhD degree. In this project, we aim to profile AML cancer cells treated with drug
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. The position is open to applicants with a PhD in computational biology, bioinformatics, biostatistics, cancer genomics, network science, or related fields. The appointment is a fulltime position and is made
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skilled doctoral researcher (PhD student) that will work in a vibrant and international research environment as part of a Research Council of Finland funded project on RNA-mediated pathogenicity mechanisms