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Field
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one of the drivers of cellular transformation and cancer development. Tumor suppressor p53 prevents proliferation of cells with damaged DNA and eliminates them either by programmed cell death or by
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lymphomas are known to be dependent on diverse microenvironmental stimuli providing them signals for survival, development, proliferation, and therapy resistance. It is known that CLL cells undergo apoptosis
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cardiomyopathy. Due to the large size of full-length plectin, classical gene-replacement strategies are not feasible, necessitating alternative therapeutic approaches. This PhD project focuses on the development
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expansion of selectively advantaged hematopoietic cells, support clonal evolution, and may contribute to malignant transformation. The dissertation focuses on understanding the molecular and genetic
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MALIGNANCIES Annotation: Transcription factors (TFs) are important regulators of cell growth, development, and hematopoietic cell differentiation. Disrupting the mechanisms that are responsible for the proper
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(or by agreement) Expected workload: 100 % (or by agreement) Job description: Leading a research team focused on the development of new methods for detection, separation and quantification of biochemically
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experimental evolution and study morphogenesis in C. elegans populations evolving in the lab. Importantly, our preliminary results show that this approach is feasible as we observed evolutionary adaptation
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recognized research teams State-of-the-art facilities Strong institutional support Opportunities for high-impact publications Career and grant-development support Initial 2-year contract Relocation assistance
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with information services. The focus of the position is on teaching, supervising final theses, and student development; it also includes collaboration on research, publications, and dissemination
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contributes to hematopoietic decline and pre-leukemic evolution, with potential relevance for early intervention strategies. Candidate profile The laboratory of hemato-oncology is searching for a highly