119 optimization-nonlinear-functions Postdoctoral positions at University of Washington
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function and produced the first ‘Boolean mouse’ to study ILC2s in vivo (Science 2024 PMID 38935708; Nature Immunol. 2023 PMID 36550322; Immunity 2023 PMID 37285842). You will be able to take advantage
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applicant will work closely with industry sponsors who will help to guide the project. The Elbert lab and the industry partner are interested in developing mechanistic, predictive models of neurodegenerative
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. The appointment will be for 1 year, with a subsequent extension to a second year depending on performance. The postdoc will work primarily with Shaffique Adam on the theory of electronic properties of twisted 2D
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candidate will investigate the functions of bile metabolites induced by bacterial infection. We aim to advance our understanding of how infection-stimulated bile metabolites influence intestinal defense
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At-Risk U.S. Communities." The postdoc will work closely with communities in flood-prone regions of the Midwest and Mississippi Delta to develop tools to predict health risks from flooding and assist local
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implement algorithms, models, and software applications that can be translated to clinical use. Job Description Primary Duties & Responsibilities: Information on being a postdoc at WashU in St. Louis can be
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, salivary gland cancers, and other tumors). We have advanced the application of single cell sequencing technologies to rigorously pursue this research, with our work providing the first detailed atlas
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, including task-based and resting state functional MRI, T1 and T2-weighted structural MRI, and diffusion MRI. The post-doc will parcellate the hippocampus and cerebellum using surface-based methods and the
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neurodevelopmental and mental health outcomes in children. This individual will have the opportunity to work with data from the Maternal and Developmental Risks from Environmental and Social Stressors (MADRES) cohort
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neurodevelopmental, neuropsychiatric, or neurodegenerative disorders, with particular emphasis on the role of mitochondrial dysfunction and metabolic deficits in disease progression. To address these questions, we use