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drug development to inhibit or deregulate disaggregase activities. We want to dissect the molecular basis of ClpG and ClpL activity control. How is the activity of the disaggregases repressed in absence
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at the Department of Gastroenterology, Hepatology and Infectious Diseases. We offer a PhD position in the highly dynamic field of “Microbiota tumor cell interaction in pancreatic adenocarcinoma (PDAC)” where we aim
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-induced adverse effects and to decipher their mechanisms. Innovative tissue engineering methods are combined with molecular biology, histology, and physiological applications to develop new approaches
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