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. Santiago Botía with close collaboration with Dr. Christoph Gerbig of the Atmosphere-biosphere Signal Attribution (BSAT) and Atmospheric measurements and mesoscale modeling (ATM) groups at the Max-Planck
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-prey contacts to understand decision making underlaying aggressive predatory biting. Using data-driven modelling, you will compare these dynamics to investigate prey choice and reward signalling. Your
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Max Planck Institute for Neurobiology of Behavior - caesar, Bonn | Bonn, Nordrhein Westfalen | Germany | about 1 month ago
behavioural data of predator-prey contacts to understand decision making underlaying aggressive predatory biting. Using data-driven modelling, you will compare these dynamics to investigate prey choice and
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of the synthetic peptide in chronic and acute heart failure animal models. The therapeutic peptide is derived from the protein S100A1, which plays a central role in maintaining normal heart function. The synthetic
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Specifications. The PhD topic consists of two parts, the weighting of which can be adjusted. First, natural frequencies of different UAV categories for different power settings shall be modelled. Second, based
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frequencies of different UAV categories for different power settings shall be modelled. Second, based on mesoscale fluid dynamic simulations, microclimatic and turbulence modelling procedures in urban
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for climate-positive and CO2-negative processes. What you will do Optimization of the pilot plant for the combustion of solid fuels (biomass, waste) with oxygen and subsequent CO2 capture Development of models
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well as Mobility Modeling and Simulation. Research topics range from conceptual issues of strategic planning to small-scale analyses of specific street spaces. The chair’s methodological focus is on the empirical
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against cardiac diseases. It is based on extensive preliminary work by our working group, which has demonstrated therapeutic effects of the synthetic peptide in chronic and acute heart failure animal models
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cell development and function, and compare the (epi)genetic profiles characteristic of individual genetic defects. These studies will be complemented by modeling disease mechanisms in suitable cell lines