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-prey contacts to understand decision making underlaying aggressive predatory biting. Using data-driven modelling, you will compare these dynamics to investigate prey choice and reward signalling. Your
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Max Planck Institute for Neurobiology of Behavior - caesar, Bonn | Bonn, Nordrhein Westfalen | Germany | about 2 months ago
behavioural data of predator-prey contacts to understand decision making underlaying aggressive predatory biting. Using data-driven modelling, you will compare these dynamics to investigate prey choice and
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of the synthetic peptide in chronic and acute heart failure animal models. The therapeutic peptide is derived from the protein S100A1, which plays a central role in maintaining normal heart function. The synthetic
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magnetic components of the dynamo are to be simulated numerically. Your profile: Potential candidates (m/w/div) should hold a Masters or Diploma degree in Materials Science, Mechanical Engineering
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well as sustainability. Its study model with Leuphana College, the Leuphana Graduate School and the Leuphana Professional School has won numerous awards. For the Institute of Management, Accounting & Finance (IMAF
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programme. Examples include stem and germ cells, biostatistics, developmental cell biology, neurosciences, and modelling and simulation of biological systems. In addition, PhD students at the DIGS-ILS have
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against cardiac diseases. It is based on extensive preliminary work by our working group, which has demonstrated therapeutic effects of the synthetic peptide in chronic and acute heart failure animal models
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University of Tübingen Neuroscience student lab Careers Back Job vacancies IT & Library / Technology & Maintenance / Administration Professorships Postdocs, assistant professors, junior research group leaders
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The Children’s University of Tübingen Neuroscience student lab Careers Back Job vacancies IT & Library / Technology & Maintenance / Administration Professorships Postdocs, assistant professors, junior research
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cell development and function, and compare the (epi)genetic profiles characteristic of individual genetic defects. These studies will be complemented by modeling disease mechanisms in suitable cell lines