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of memory, senescent, and exhausted T-cells with age. The PhD candidates will work on: Identifying key metabolic pathways involved in T-cell aging using advanced genetic models that allow for parallel CRISPR
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events in model systems revealed the existence of diverse regulatory principles, however, the mechanisms by which rapid and coordinated changes in the AS output can be achieved are still poorly understood
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of this project is on the role of APCs in experimental autoimmune encephalomyelitis (EAE), a mouse model for the human autoimmune disease of the central nervous system (CNS) multiple sclerosis (MS). In EAE
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: Send your full application in one pdf with a short statement of research interests, CV, certificates, or transcript of records. Contact:
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finds and invades the sister chromatid (or homologous chromosome) to use it as template to copy and recover the broken DNA sequence. In comparison to HR, NHEJ is “error-prone” as it frequently requires
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chemically induced protein degradation. This will allow us to model developmental diseases and unravel the physiological role of BAF subunits and specific BAF complexes in human developmental processes. With
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detailed application with the usual documents quoting the reference number “w24‑215” by June 6, 2025 (stamped arrival date of the university central mail service or the time stamp on the email server of TUD
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“w25-125” by June 6, 2025 (stamped arrival date of the university central mail service or the time stamp on the email server of TUD applies) to: TU Dresden, Chair of Inorganic Molecular Chemistry, Prof
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arrival date of the university central mail service or the time stamp on the email server of TUD applies), preferably via the TUD SecureMail Portal https://securemail.tu-dresden.de by sending it as a
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(DAAD) Branch Office Beijing DRC Building D1, 1102A 19 Dongfang Donglu, Chaoyang District 100600 Beijing, VR China Tel.: +86 010/6590 6656 Fax.: +86 (10)/6590-6393 E-Mail: postmaster@daad.org.cn WWW