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the molecular mechanisms driving therapy resistance in human cancers. The research project focuses on understanding the molecular mechanisms by which MAPK4 and/or COP1 regulate cancer progression and therapy
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Neurological Research Institute, Texas Children’s Hospital. Immediate position available at the level of Postdoctoral Associate to study the mechanisms underlying astrocyte-tumor interactions. The focus
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models, human tissue, stem cell models, and single-cell multi-omics to advance precision medicine. We place a particular emphasis on understanding mechanisms of cardiac arrhythmias to identify more
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, genetic, and immunological approaches to understand the mechanisms of cancer predisposition, initiation, and progression and to develop agents (compounds, antibodies, and vaccines). The postdocs will
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groups pioneered human organoid models to study host-virus interactions, and ongoing work is expanding these systems to include microbial communities. The ultimate goal is to uncover mechanisms and
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enormous genetic information to functional information). We place emphasis in "Translational Research": from “discovering mechanisms” to “developing innovative treatment strategies” to “moving into clinical
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to join our dynamic research team investigating post-transcriptional and epigenetic mechanisms that control cell fate, stem cell plasticity, and cancer progression. Research Focus We employ cutting
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Biology in Cancer Genomics and Immunology. This position will be involved in the development and/or application of computational approaches to understand the mechanism of cancer development, progression
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devoted to unraveling the mechanisms of how central nervous system orchestrates systemic energy balance, aiming to unlock groundbreaking therapies for obesity and metabolic disorders. The research projects
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analytical and problem-solving skills. This position offers an opportunity to work on NIH-funded projects investigating the immunologic mechanisms underlying chronic lung allograft dysfunction, with a focus on