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understanding the risk mechanisms that underlie the differing profiles (and course) of depression to inform more targeted approaches to prevention and treatment efforts. Emerging evidence suggests that weaknesses
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/molecular interactions between stromal-innate immune cells involved in shaping the pro-inflammatory response in the joint. This will give significant insight into the (i) differential pathogenic mechanisms
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nanomaterials. This will be achieved by developing and codifying DES-based routes to key classes of environmentally relevant nanomaterials, including (photo)electrocatalytic metal oxides; phosphate-based
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