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Posting Details Position Information Fiscal Year 2025-2026 Position Title Postdoctoral Associate, Microbiology and Immunology Classification Title Postdoctoral Associate Department Microbiology and
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Applications are invited for a temporary post of a Post-doctoral Research Fellow Level 1 or 2 within UCD Sutherland School of Law Prior to application, further information (including application
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Term: 1 year Appointment Start Date: TBD Group or Departmental Website: https://yangresearchlab.stanford.edu/ (link is external) https://med.stanford.edu/microimmuno.html (link is external) https
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positions are pro-rated accordingly. The University also offers a comprehensive benefit program to eligible employees. Please see this link for more information. Requisition No: D-26-PSY-00002 PI278656689
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. The University also offers a comprehensive benefit program to eligible employees. Please see this link for more information. Requisition No: D-26-PPH-00001 PI278657033 Create a Job Match for Similar Jobs About
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infrastructure that enables secure, large-scale data integration across Europe. The postdoc will focus on two pillars: Multi-omics data integration: developing robust pipelines and frameworks for preprocessing
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LanguagesENGLISHLevelExcellent Research FieldGeography Additional Information Additional comments Reference: 2025DILIFRUA118 You will find the bases of the call in the following link: https://tauler.seu-e.cat/detall?idEns=11
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upload the required Authorization of Information Release to your application. Examples of developed computer code, link to source code repository, or description of developed proprietary code (Optional
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10 Oct 2025 Job Information Organisation/Company Max Planck Institute for Psycholinguistics Research Field Computer science » Programming Computer science » 3 D modelling Researcher Profile
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The University of North Carolina at Chapel Hill | Chapel Hill, North Carolina | United States | about 1 hour ago
to uncover mechanistic links between the genome (e.g., DNA sequence variants), epigenome (chromatin accessibility), gene expression, and air pollutant-induced airway inflammation phenotypes in a mouse model