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: From Mechanisms to Therapies”. In this CRC we aim to understand how plasticity i.e. non-genetic adaptations of the bone marrow niche induced by the disease or therapeutic agents contribute to drug
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“Multi-modal proteomics to understand and overcome niche-related adaptive drug-resistance mechanisms in FLT3-mutated AML“ we are looking for a PhD student. The project is is part of the SFB/CRC 1709
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mechanistic and clinical studies. To expand our activities, we are looking for a motivated PhD student to investigate mechanisms of disease and drug resistance in myeloid leukemia. Job-ID: V000014484 Field
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“Cellular Plasticity in Myeloid Malignancies: From Mechanisms to Therapies”. In this CRC we will focus on myeloid malignancies as a model to dissect the various molecular mechanisms that enable and regulate
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Malignancies: From Mechanisms to Therapies”. In this CRC we will focus on myeloid malignancies as a model to dissect the various molecular mechanisms that enable and regulate cancer cell plasticity in AML. Our
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, and therapy resistance mechanisms Ability to work independently and collaboratively within interdisciplinary teams Prior experience with network modeling or machine learning is a plus We offer
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nanobelts. Your profile: We are looking for experimentally skilled candidates, ideally experienced in multiple-step organic synthesis Prior work on fused aromatic compounds is certainly advantageous
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the ability to work both independently and collaboratively. Experience in animal experimentation is a plus. Enthusiasm for onco-immunology and microbiome research and the drive to contribute to scientific
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experimental therapy of myocardial insufficiency. The aim of the project is to develop additional cell-permeable S100A1ct peptidomimetics with extended therapeutic capabilities and investigate
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muscle and, due to its pharmacokinetic and pharmacodynamic properties, can be used and further developed as a candidate for the experimental therapy of myocardial insufficiency. The aim of the project is