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deformities ultimately leaving them severely disabled. This lifelong disorder is currently incurable. The most prominent protein family implicated in the etiology of CMT are aminoacyl-tRNA synthetases (ARS
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sequencing and transcriptomics, and iPSC-based disease modeling to better understand the genetic etiologies of intractable epilepsy. The team is part of the European STXBP1 consortium . About the project
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diverse academic backgrounds to contribute to our projects in areas such as: Network Security, Information Assurance, Model-driven Security, Cloud Computing, Cryptography, Satellite Systems, Vehicular
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reconstruction framework for reproducible estimation of the cerebral blood flow in the mouse brain. Once established, the framework will be applied in animal models of Huntington’s Disease. You will publish
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weakness and wasting, sensory loss, and skeletal deformities ultimately leaving them severely disabled. This lifelong disorder is currently incurable. The largest protein family implicated in the etiology
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diverse academic backgrounds to contribute to our projects in areas such as: Network Security, Information Assurance, Model-driven Security, Cloud Computing, Cryptography, Satellite Systems, Vehicular
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on cancer metastasis and novel metabolic pathways. We exploit mouse models, genetic engineering, metabolomics and single cell & spatial multi-omics analysis to gain groundbreaking insights into metabolism as
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on cancer metastasis and novel metabolic pathways. We exploit mouse models, genetic engineering, metabolomics and single cell & spatial multi-omics analysis to gain groundbreaking insights into metabolism as
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malignancy and is the second leading cause of cancer-related deaths worldwide. Current treatment options such as chemotherapy and targeted/immunotherapies are insufficient due to resistance development and
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short and long-read sequencing and transcriptomics, and iPSC-based disease modeling to better understand the genetic etiologies of intractable epilepsy. The team is part of the European STXBP1 consortium