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these interactions modulate neural circuit function in health and disease. We are seeking a highly motivated individual to study mechanisms of how T cells modulate resident brain cells during neuroinflammation
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of Traumatic Brain Injury, particularly as it applies to biomarkers, including long-term follow-up of humans and related future neurodegenerative disease. Applicants must have a Ph.D. degree. Research
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. The candidate will also have an interest in neuropsychology and clinical psychology, preferably with a specific interest in childhood encephalitis and acquired brain injury. The PhD studentship (co-funded by
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. Recent findings have identified complement activation in tuberous sclerosis complex (TSC)—a rare genetic disorder caused by mutations in TSC1 or TSC2, leading to hyperactivation of the mTOR pathway and a
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brain activity patterns and transforms them into actionable commands, enabling direct communication between the brain and external devices. BCIs have broad applications, from restoring movement in people
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Introduction Neurodegenerative diseases, such as Alzheimer’s disease (AD) and Parkinson’s disease (PD), affect tens of millions of people worldwide and cause a heavy economic burden on societies
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for brain development and is closely linked to neurodevelopmental disorders such as anxiety and depression. While serotonergic axons reach most forebrain regions by birth, their maturation involves region
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The overarching aim of the project is to understand how early neurodevelopmental risk factors disrupt brain maturation and contribute to psychosis vulnerability across development. This PhD project
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the gut-brain and gut-muscle axes, as potentially key modulators of cognitive function, and physical health in middle-aged-to-older adults. The central theme will be to investigate how targeted
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– i.e., the light, volume and pitch changes from which we extract meaning – has increased continuously since we have been producing it. Our brains work by generating and testing predictions – but younger